Optic ataxia entities

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Revision as of 03:01, 21 October 2022 by Oldver>Mehrdad (Created page with "Optic ataxia is the lack of coordination between visual input and hand movements. For example, the patients can accurately touch their own body voluntarily, but when given a visually guided task, they are unable to complete it. However, once provided proprioceptive or auditory cues to reach for a target object, they can perform the task smoothly. Theories are that this deficit may arise due to damage to the superior parietal lobule, which is responsible for coordination...")
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Optic ataxia is the lack of coordination between visual input and hand movements. For example, the patients can accurately touch their own body voluntarily, but when given a visually guided task, they are unable to complete it. However, once provided proprioceptive or auditory cues to reach for a target object, they can perform the task smoothly. Theories are that this deficit may arise due to damage to the superior parietal lobule, which is responsible for coordination between visual input and hand movements.[1] Isolated optic ataxia may occur from lesions in Broadman’s areas 5, 7, 19, 37, and 39. [2] Although existing literature suggests that parietal and occipital lobes are the primary areas of involvement in Balint syndrome, lesions in other areas of the brain such as the prefrontal cortex and frontal eye fields were reported to be associated with individual symptoms of Balint syndrome.[2]

  1. Amalnath SD, Kumar S, Deepanjali S, Dutta TK. Balint syndrome. Ann Indian Acad Neurol. 2014 Jan;17(1):10-1.
  2. 2.0 2.1 Rizzo M, Vecera SP. Psychoanatomical substrates of Bálint's syndrome. J Neurol Neurosurg Psychiatry. 2002 Feb;72(2):162-78.