Nerocognitive considerations: Difference between revisions
Oldver>Mehrdad |
m (1 revision imported) |
(No difference)
|
Revision as of 03:04, 23 March 2023
There is no doubt that Nerocognitive area is the most complex area of diseases in the body. Apart from the incomplete information of neuroscience, the network function of neurons should be considered as the solution of evolution to provide the most economical organ of the body. This means that a neuron, by creating a network of 10,000 neurons around itself, sometimes participates in several complex brain functions at the same time, and this challenges the simple mind of Newtonian scientists.
Since the goal of TLT theory is to provide a solution to simplify the understanding of disease patterns, and this goal was met with significant success in pilot studies on other body organs except the brain, I was encouraged to implement this model on the brain as well. Which, of course, was a wonderful dream that is gradually turning into a deadly nightmare. Until I realized that there are special rules in the brain that make it dependent on other organs. It is better to say that it may not be possible to claim with certainty that such laws are inherent in the brain, but these laws can be included in brain modeling to simplify and understand the controversy in the brain.
The first thing that comes to the mind of every viewer is the multiple dimensions of brain activity.
Movement - Sense - Cognition - Emotion - perception
This area is well known in neuroscience and I will use it as much as possible, of course, on the condition that I organize my toolbox first.
The next area is the quantification of brain function. The key question in any brain disorder is whether this disorder causes a decrease or an increase in brain function. Obviously, this ruler can be used in all the areas mentioned above
Thus, with these two tools, I will be able to classify all brain diseases in a simple way. For example, depression slows down everything, especially in the field of emotion, perception, cognitive functions, etc., and the difference with catatonia is that catatonia obviously slows down the motor system and Depression leads the emotional system and hypomania leads the patient to the rhythmic increase and decrease of the emotional system
Functional oriented study
There are different ways to classify the disease. Classification based on etiology, structural disorder, histologic deviation or pathology and functional classification. The ultimate goal is that these categories eventually reach the same conclusion. For example, in pharyngitis, pharyngeal tissue is inflamed and inflammatory cells can be seen in sampling. The structure of the tissue changes macroscopically and becomes covered with exudative secretion. Its function in swallowing and breathing is also disturbed, and this becomes a clean disease called pharyngitis
This happens in most diseases except brain diseases. Brain has another story. Of course, the ideal is what was said, but this goal still seems unattainable. Moreover, the etiology of most brain diseases is still unclear. The symptoms of brain diseases overlap a lot, and the course of symptoms is so variable that it is difficult to imagine a clear rule for them. One way is to tell patients to wait until the exact cause of their illness is determined. But since no one has the patience of Job, it is necessary to start the treatment of brain diseases with this little wealth.
Today, most brain diseases are diagnosed in the clinical field rather than depending on para clinical diagnosis. Neuropsychologists are not waiting for para clinical documentation to start treatment
This means that brain function takes precedence over its structure. If we add the Compensation ability of brain parts which is now known as neuroplacticity, we simply give the right to this view.
The second point is the emphasis of TLT theory on the dynamics of diseases. In fact, according to the definition of miasm, the repeating pattern is the function of the disease. Although the role of histological destruction in occurrence of diseases is not deniable, what is observed by the miasm and considered as the goal of treatment is not the change of the tissue structure, but the improvement of its function.
These are two good reasons why I prefer a functional approach to categorizing brain diseases.
Central | Peripheral | ||
---|---|---|---|
Cranial nerves palsy
|
Trigeminal neuralgia
Diplopia (3, 4, 6) Bels palsy |
||
Cortical Blindness | Peripheral Blindness | ||
Central labyrintitis (Hearing + Ataxia) | only ataxia | ||
Anosemia | Coryza, Rhinitis | ||
/ |
Decreased | Increased | Changeable | |
---|---|---|---|
Cognition impaired | |||
Perception | Impaired | Hallucination | |
Thought | Dementia | Delusion | |
Memory / Concentration | Impaired | Anxiety / Obsession | |
Mood | Depression | Agitation / Anger/ Fear | Changeable |
Peripheral | Central | ||
---|---|---|---|
Decreased | Hypostasia | ||
Increased | Hyper stasia | Central sensitization | |
Deviared | Dysesthesia
Pruritis |
LMN | UMN | Central | |
---|---|---|---|
Decreased | LMN | Brady kinesia | |
Increased | Myoclonous
Clonous Jerking / Twitching
|
Hyper kinetics | |
Rhythmic | Non-Rhythmic | ||
---|---|---|---|
Simple | Tremor | ||
Complex | Chorea / Sterotypia / Preservation / Tic/ | ||
Peripheral | |||
Central | Rigidity |
Reflexes | Tonocity | Hyper kinetics | Muscular bulk | |
---|---|---|---|---|
UMN | Hyper reflexia | Spasticity
Hypertonia |
Clonus | --- |
LMN | Hypo reflexia | Hypotonia | Fasciculations | Atrophy |
Hyper | |||
---|---|---|---|
Central | |||
Peripheral | LMN | ||
UMN |