NVCJD, New Variant Creutzfeldt Jakob Disease: Difference between revisions
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Revision as of 03:04, 23 March 2023
Core
NVCJD focuses on Pulvinar nucleus of Thalamus
- Visual attention / Impaired concentration
- Visual salience
- Social cognition
- Neglect syndrome / Disorientation = Spatial Confusion
- Gait disorder /Incoordination
Entities
UMNS
- Central inhibitory control (pyramidal) has been removed from the spinal cord
- Hypertonia (Possibly equals to Spasticity)
- Myoclonus
- Hyperreflexia
- Primitive reflexes
Psychosis
- Auditory / Visual Hallucination
- Delusions
- Behavioral change
- Agitation, Irritability, Anger, Sleeplessness
- Dysesthesia , Painful Paresthesia
Ataxia (Maybe)
- Incoordination
- Gait abnormalities
Autonomic dysfunction (Maybe)
- Recent researches suggest that vCJD attack brain from the GI tract autonomic nervous system (Both parasympathetic and sympathetic)[1]
- Dysarthria (Possibly UMN Dysarthria)
- Dysphagia
- Urine Incontinence
Choreathetosis
Diseases
Dementia
- Disorientation
- Impaired Cognition
- Memory; weakness
- Depression
- Anxiety
Schizophrenia
- Psychosis
- Hallucination, Auditory, Visual
- Delusion
- Altered though pattern
- Behavioral change
Remedies
- LACH, BELL
- Stram
- Crot-c, Carbn-s
- ↑ Amanda L. Woerman, Gültekin Tamgüney,↵↵Body-first Parkinson’s disease and variant Creutzfeldt–Jakob disease – similar or different?,↵↵Neurobiology of Disease,Volume 164, 2022,