Disinhibition: Difference between revisions

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=== Clinic ===
=== Clinic ===


* It is a lack of restraint manifested in disregard of social conventions, impulsivity, and poor risk assessment.  
* It is a lack of restraint manifested in disregard of social conventions, [[impulsivity]], and poor risk assessment.  
* Disinhibition affects motor, instinctual, emotional, cognitive, and perceptual aspects with signs and symptoms similar to the diagnostic criteria for mania
* Disinhibition affects motor, instinctual, emotional, cognitive, and perceptual aspects with signs and symptoms similar to the diagnostic criteria for [[Mania remedies|mania]]
* Hypersexuality, hyperphagia, and aggressive outbursts are indicative of disinhibited instinctual drives.
* [[Sexual desire increased remedies|Hypersexuality]], [[hyperphagia]], and aggressive outbursts are indicative of disinhibited instinctual drives.
* It range from mildly inappropriate social behavior, lack of control over one's behavior to the full-blown mania
* Behavioral changes would be its best synonym in viral miasms.




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It is an entity with these signs and symptoms
It is an entity with these signs and symptoms


* Impulsivity
* [[Impulsivity]]
* Disregard for others and social norms
* '''personality changes:''' Disregard for others and social norms
* Aggressive outbursts
* Aggressive outbursts
* Misconduct and oppositional behaviours
* Misconduct and oppositional behaviors
* Disinhibited instinctual drives including risk taking behaviours and hypersexuality.  
* Disinhibited instinctual drives including risk taking behaviors and hypersexuality.
* Diffuse cognitive impairment, with more prominent deficits in the rate of Information processing, Attention, memory, Cognitive flexibility, and Problem solving.




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=== Causes ===
=== Causes ===


* Disinhibition is a common symptom following brain injury, or lesions, particularly to the frontal lobe and primarily to the orbitofrontal cortex.  
* TBI (Traumatic Brain Injury) or lesions, particularly to the frontal lobe and primarily to the orbitofrontal cortex.
* The neuropsychiatric sequelae following brain injuries could include diffuse cognitive impairment, with more prominent deficits in the rate of information processing, attention, memory, cognitive flexibility, and problem solving.
* The neuropsychiatric sequelae following brain injuries could include  
*Prominent impulsivity, affective instability, and disinhibition are seen frequently, secondary to injury to frontal, temporal, and limbic areas. In association with the typical cognitive deficits, these sequelae characterise the frequently noted "'''personality changes'''" in TBI (Traumatic Brain Injury) patients.
*Tumors / Strokes / Epilepsy
* Disinhibition syndromes, in brain Injuries/ Tumors / Strokes / Epilepsy  




=== Related entities ===
=== Related entities ===


* It range from mildly inappropriate social behaviour, lack of control over one's behaviour to the full-blown mania
* Cognitive impairments such as
* Several studies in brain traumas and insults have demonstrated significant associations between disinhibition syndromes and visuospatial functions, somatosensation, and spatial memory, motoric, instinctive, affective, and intellectual behaviours.
** Visuospatial functions
 
** Somatosensation
=== Entity ===
** Spatial memory
Behavioral changes would be the best synonym in viral miasms.
* Impaired motoric, instinctive, affective, and intellectual behaviors.
 
 
 


=== Related Diseases ===
=== Related Diseases ===
It have also been reported with
* Mania-like manifestations in old age with lesions to the orbito-frontal and basotemporal cortex involving limbic and frontal connections (orbitofrontal circuit), especially in the right hemisphere.
* Mania-like manifestations in old age with lesions to the orbito-frontal and basotemporal cortex involving limbic and frontal connections (orbitofrontal circuit), especially in the right hemisphere.
* Alcohol consumption
* [[ADHD]], hyperactive/impulsive subtype
* CNS depressants drugs, e.g., benzodiazepines that disinhibit the frontal cortex from self-regulation and control.
* ADHD, hyperactive/impulsive subtype
* Conduct disorder
* Conduct disorder
* Anti-social personality disorder
* Anti-social personality disorder
* Substance abuse
* [[Substance use disorder|Substance abuse]] / Alcohol consumption
* CNS depressants drugs, e.g., benzodiazepines that disinhibit the frontal cortex from self-regulation and control.

Latest revision as of 01:29, 26 September 2023

Clinic

  • It is a lack of restraint manifested in disregard of social conventions, impulsivity, and poor risk assessment.
  • Disinhibition affects motor, instinctual, emotional, cognitive, and perceptual aspects with signs and symptoms similar to the diagnostic criteria for mania
  • Hypersexuality, hyperphagia, and aggressive outbursts are indicative of disinhibited instinctual drives.
  • It range from mildly inappropriate social behavior, lack of control over one's behavior to the full-blown mania
  • Behavioral changes would be its best synonym in viral miasms.


Signs / Symptoms

It is an entity with these signs and symptoms

  • Impulsivity
  • personality changes: Disregard for others and social norms
  • Aggressive outbursts
  • Misconduct and oppositional behaviors
  • Disinhibited instinctual drives including risk taking behaviors and hypersexuality.
  • Diffuse cognitive impairment, with more prominent deficits in the rate of Information processing, Attention, memory, Cognitive flexibility, and Problem solving.


Causes

  • TBI (Traumatic Brain Injury) or lesions, particularly to the frontal lobe and primarily to the orbitofrontal cortex.
  • The neuropsychiatric sequelae following brain injuries could include
  • Tumors / Strokes / Epilepsy


Related entities

  • Cognitive impairments such as
    • Visuospatial functions
    • Somatosensation
    • Spatial memory
  • Impaired motoric, instinctive, affective, and intellectual behaviors.

Related Diseases

  • Mania-like manifestations in old age with lesions to the orbito-frontal and basotemporal cortex involving limbic and frontal connections (orbitofrontal circuit), especially in the right hemisphere.
  • ADHD, hyperactive/impulsive subtype
  • Conduct disorder
  • Anti-social personality disorder
  • Substance abuse / Alcohol consumption
  • CNS depressants drugs, e.g., benzodiazepines that disinhibit the frontal cortex from self-regulation and control.
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